Lyme Disease

In parts of the United States, the most common tick-borne disease is Lyme disease. This emerging infectious disease is caused by a spirochetal (spiral-shaped) bacterium, Borrelia burgdorferi. Lyme disease usually is treated successfully in the early stages with antibiotics. Patients who go untreated or who do not respond to antibiotics may develop a chronic multi-system disease with an unpredictable array of symptoms. Many of these symptoms mimic those of other diseases.

Chronic Lyme disease most often produces persistent arthritis or nervous system problems, although the heart also can be involved. Lyme arthritis usually affects one or several large joints, often the knee. If the central nervous system is involved, symptoms may include headaches, nausea and vomiting, memory loss and a variety of other cognitive, behavioral and sleep problems. Involvement of the peripheral nerves can result in radiating pain in the limbs, numbness and partial paralysis.

No one knows why in some patients with late Lyme disease symptoms eventually diminish or disappear, whereas in other patients the symptoms persist. Scientists think that in some cases the spirochete may evade the immune system. It then survives in numbers too low to be detected by conventional tests, yet high enough to produce illness. Some scientists speculate that ongoing infection may even be caused by a second tick-borne pathogen. Persistent symptoms also may be the result of an overactive immune response that continues to injure the host?s tissues long after the organism has been eradicated.

Continued research is essential to making progress against this disease. Since 1981, when NIAID scientists first isolated the responsible organism, the Institute has supported an active research program on Lyme disease. Much of this research focuses on the pathogenesis, or disease process. This includes the study of the biology of B. burgdorferi, how it evades the immune system, how it interacts with its human host, its genetic components that allow the organism to control surface protein expression, and differences in human genes that account for the variations in the immune response among individuals.

There is no uniformity among laboratories that perform tests to detect antibodies in the serum of the blood, contributing to the misdiagnosis of Lyme disease. To overcome this problem, NIAID staff met with officials of the Centers for Disease Control and Prevention (CDC) in the fall of 1994 to discuss standardization of the Western Blot diagnostic test. Guidelines for laboratories that perform and interpret serologic tests were developed and a summary was published in the CDC?s Morbidity and Mortality Weekly Report (MMWR). Further improvements in existing tests, as well as the development of new technologies to diagnose Lyme disease remain an Institute priority.

Since 1994, NIAID has convened meetings to address the issues surrounding chronic Lyme disease. Attending were scientists involved in Lyme disease at NIH and elsewhere, physicians and patient advocates. The participants acknowledged that determining whether chronic Lyme disease is caused by persistent infection or is a post-infectious disorder is a major research goal. Finding the answer to this question for any individual patient will have an important bearing on his or her treatment. While the participants acknowledged the difficulties in carrying out clinical trials to evaluate chronic Lyme disease, they agreed that clinical trials are necessary to resolve questions about optimal treatment.

Participants agreed that the first trial should focus on a well-defined patient population with probable B. burgdorferi infection that might respond to antibiotics. Patients could then be selected on the basis of relapse or non-response following appropriate treatment for early-stage Lyme disease. This would provide common criteria for studying and treating this multi-symptom disease. Such patients might include (1) those with persistent arthritis or persistent fatigue or fibromyalgia; (2) those with cognitive abnormalities, neuroradiculitis, headache or encephalomyelitis; and (3) those with objective evidence of continuing B. burgdorferi infection.

The group discussed possible clinical trial designs for assessing the effectiveness of antibiotic therapy for the treatment of chronic Lyme disease. A Request for Proposals (RFP) reflecting this discussion was issued to solicit proposals for conducting placebo-controlled studies in that regard. After rigorous review of all proposals submitted, this five-year initiative is now under way and involves several collaborating institutions. Another closely related study also is being funded by means of a research grant. Additionally, NIAID and other NIH scientists have launched a separate intramural study to better characterize and treat ongoing Lyme disease. Researchers from both studies will work closely together with the NIAID Project Officer and under the guidance of an advisory panel of Lyme disease scientists, medical experts, and patient representatives.

Meanwhile, NIAID continues to pursue the underlying mechanisms of B. burgdorferi infection through grants to study immune response to Lyme disease infection and vaccination, and contracts to study animal models of chronic Lyme disease. These efforts will ultimately advance our understanding of chronic Lyme disease and lay the groundwork for future clinical trials.

Also see fibromyalgia and lyme disease

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