Trypanosomiasis
“The kiss
of death”
Introduction
American
Trypanosomiasis, also known as Chagas
Disease, was discovered by Carlos Chagas. In 1909, while working
for the
Oswaldo Cruz Institute, Chagas was sent to Lassance, Brazil to help
fight an outbreak
of malaria. While living there, he observed that the rural homes
were
infested with “kissing bugs,” and that these bugs bit and sucked blood
from
faces of people while they were sleeping. He decided one day to
inspect
the inside of the intestines of the bugs. Upon doing so he found a new
species
of flagellate protozoa. He proved experimentally that monkeys
could get
this parasite by being bitten by an infected bug. Because these
bugs were
found in people’s homes and biting them, Chagas suspected that this
parasite
caused disease in humans as well. On April 23, 1909, he first
discovered
the parasite in the blood of a sick three-year-old girl, proving that
it caused
disease.
Transmission
Aspects
The
disease is found
in Mexico, Central American, and Northern South America. The causative
agent, a
protozoan parasite, Trypanosoma cruzi, enters the body through
an open
wound or mucous membrane. The wound is normally caused by the
bite of a
revuiid bug, such as Triatoma infestans, Rhodnius polixus, and
Panstrongylus
megistus.
The
revuiids are found in rural areas, in the cracks of poorly
constructed homes. Also, they are found in homes made from mud
and
thatch. People who sleep in these kinds of homes are the most
likely to
become infected. These bugs especially like to bite children on
their
faces, and are therefore known as “kissing bugs.”
The
disease is transmitted a few different ways:
The bugs
become infected
after biting and sucking on the blood of an infected human or other
animal. A human that becomes infected when an open wound or
mucous
membrane comes in contact with the feces of these bugs. The feces
contain
the parasites, which are in the metacyclic trypomastigotes stage of
their
lives. Once inside the bloodstream, they penetrate various
tissues.
Inside these cells the metacyclic trypomastigotes transform to
amastigotes. Within the infected cells, the amastigotes divide by
binary
fission. The amastigotes once again become trypomastigotes, and
then are
released from the cells, destroying them. At this point, the
trypomastigotes can either infect other cells within the host or become
amastigotes again, or they are ingested by a revuiid bug. If
ingested,
the trypomastigotes develop into epimastigotes. They multiply in
the gut
and then move to the hindgut, where they develop into metacyclic
trypomastigotes. At this point, they leave the host in its feces
and then
are free to infect other humans and animals.
There are
no misconceptions of the spread of
this disease. Some people may think that just by staying in
areas/countries where the bugs are found, they could get the disease.
They have
to stay in poorly constructed homes, this is where the bugs are
founds.
Tourists are safe when they stay in hotels.
Damage
Done to Host
Types of
Damage:
The
parasite T. cruzi plays a major
role in creating organ lesions in the host, usually more commonly and
intensively in the heart, esophagus and the colon. These lesions lead
to an
inflammatory response, cellular lesions and fibrosis.
Causes of
Damage:
Trypansomas
cruzi, the
parasite
involved in Chagas disease. It has four forms and only one is
parasitic, the
trypomastigote. The blood trypomastigote is an infective flagellate
found in
the blood and the metacyclic trypomastigote is found in the terminal
part of
the digestive and urinary tracts.
As with
the transmission of the disease, there are no common misconceptions on
how the
disease is presented, except for how exactly the disease is contracted.
The
bugs themselves do not have the disease until they are infected with
contaminated feces.
Clinical
Characteristics
Signs and
Symptoms
There are
three stages of Chagas disease, the acute stage,
intermediate stage, and the chronic stage, with each stage presenting
different
symptoms. The acute stage of the disease can occur within a few days to
a few
weeks of the initial bite. In the acute stage of Chagas the patients
generally
have a distinct swelling on one eye, called Romaña’s sign, other
symptoms in
this stage include:
The
intermediate stage of the disease presents no symptoms and this
stage occurs 8 – 10 weeks after infection and can last for many years
before
the chronic symptoms manifest. The chronic stage occurs years after the
initial
infection (10 – 30 years), and these symptoms are more devastating than
that of
the acute stage and they include:
Diagnosis
of Chagas
Disease
During
the acute stage of the disease the motile trypanosomes
parasite
can be detected by blood smears. To detect the parasite there are
traditional
methods that can be used including:
Though
the above tests are highly specific
they are very time consuming and have a low sensitivity. A more
sensitive test
that has proven to be a better alternative is PCR (polymerase chain
reaction).
The PCR technique is used to amplify the kinetoplast DNA of the trypanosomes,
and it is a good detector of Trypanosoma
cruzi. The best
method to
diagnose the chronic or intermediate stage of the test is by using
serological
test using the antibodies against the parasite. The serological test
includes:
The
problem with some of these test are that the results can be
inconsistent, so a negative result does not actually mean that the
person is
free of infection. This is due to the low sensitivity of the test used.
The
serological test such as the ELISA, or IFA would show a positive result
even
though the parasitology test was negative. There should be more test
performed
for Chagas detection because some say that the serological test are
accurate
and other say that they are not.
Once the disease has been diagnosed the appropriate course of treatment
includes Benznidazole.
Once the disease has proceeded
to the chronic stage antibiotics are effective in treatment. There is
currently
no available vaccine for the disease.
References
Chiari Egler. Chagas Disease Diagnosis Using Polymerase Chain Reaction, Hemoculture and Serologic Methods. 1999; 299 - 300
Walter M. R. Oelemann, Maria Da Glória M. Teixeira, Giovani C. Veríssimo Da Costa,José Borges-Pereira, José Adail F. De Castro, José Rodrigues Coura, and José Mauro Peralta Evaluation of Three Commercial Enzyme-Linked Immunosorbent Assays for Diagnosis of Chagas ‘disease. 1998; 2423-2427
Murray, Patrick. Ken Rosenthal and Michael Pfaller. Medical Microbiology. 5th Ed. Elsevier Inc. 2005.
Kirchhoff LV. American trypanosomiasis (Chagas' disease)--a tropical disease now in the United States. New England Journal of Medicine. Vol. 329:639-644, August 26, 1993
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Contact
Info:
Bina Dharia
Jide Fagbemi
Ellen Valley